A growing body of evidence shows that susceptibility to adult-onset CVD begins in early life, even in utero, presumably as a result of epigenetic dysregulation.
A growing body of evidence shows that susceptibility to adult-onset CVD begins in early life, even in utero, presumably as a result of epigenetic dysregulation.
Over time, increased hemodynamic stress on the heart from high blood pressure leads to the compensatory development of left ventricular hypertrophy.
Mounting evidence suggests that some viruses can persist in heart tissue after an initial infection, and become reactivated under certain conditions, triggering a cell-mediated immune response that leads to myocarditis and, potentially, dilated cardiomyopathy.
Preliminary findings suggest that the nutritional supplement combination might be clinically effective in reducing fasting glucose and A1c in prediabetic adults and those with type 2 diabetes.